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Exploiting DNA Replicative Stress for Novel Small Cell Lung Cancer Therapies — Anish Thomas (DIVISION OF BASIC SCIENCES - NCI)

1. Targeting replication stress in cancer: Stalled replication forks activate ataxia telangiectasia and Rad3-related kinase (ATR), which subsequently phosphorylate downstream kinases including CHK1. T

Project titleExploiting DNA Replicative Stress for Novel Small Cell Lung Cancer Therapies
Project number1ZIABC011793-06
PI nameAnish Thomas
InstitutionDIVISION OF BASIC SCIENCES - NCI
Funding mechanismZIA
AgencyNCI
Award amount$2.22M
Fiscal year2023
Abstract1. Targeting replication stress in cancer: Stalled replication forks activate ataxia telangiectasia and Rad3-related kinase (ATR), which subsequently phosphorylate downstream kinases including CHK1. T
Key terms<ACSL1><ACSL1 Gene><ASCL1><ASCL1 gene><ASCL1 protein><ASH1><ATR gene><ATR protein><ATR protein kinas

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